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Inflammatory substances in the muscle produce pain by sensitizing nerve endings and receptors
Positively and negatively enriched pathways were identified separately using default parameters, including g_SCS correction
Therefore, the NF-B signaling pathway and subsequent glucose metabolic adaptation could be a promising target for rectifying the abnormality of PDAC energy metabolism
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Repurposing ezetimibe as a neuroprotective agent in a rotenone-induced Parkinsons disease model in rats: Role of AMPK/SIRT-1/PGC-1 signaling and autophagy
The spectrum of psychiatric symptoms in Wilsons disease: treatment and prognostic considerations
