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Our understanding of the means by which GH and IGF-1 regulate growth was set into motion after experiments conducted in 1957 by Salmon and Daughaday showed that a factor in the serum (sulfation factor) was able to stimulate sulfate incorporation into cartilage in vitro [137]

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In addition to the role of HDL in stabilizing plaques, recent studies have focused on the lesional loss of specialized proresolving mediators (SPM) versus proinflammatory factors (i.e

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The cytokine TGF- triggers the Janus kinases (JAKs) / signal transducer and activator of transcription (STAT) proteins (JAK/STAT) pathway in certain contexts, while suppressing it in others [49]