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Notably, the two-dimensional ultrathin structure (thickness ~2.5 nm) further shortened ion diffusion pathways, enabling capacitive contribution to reach 88.05% at high scan rates

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Taken together, these data suggest that LA regulates tight junction assembly in leaky epithelia in part via a mechanism related to regulation of MLC phosphorylation

However, despite molecular evidence of oxidative damage, antioxidant therapies remain underutilized in the clinical management of obesity-related endometrial disorders

To summarize, CAFs and ECM within the TME influence the HGF/c-MET signaling pathway through their interactions with immune cells and the secretion of HGF, leading to the formation of an immunosuppressive TME that accelerates tumor growth

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