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reported that IFN--primed human MCs caused abundant immunologic synapses with CD4 + T cells, concomitantly with an enhancement of the production of Th22 and IL-22/IFN--producing Th cells from the circulating memory CD4 + T-cell pool

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Disrupting the immune homeostasis: the emerging role of macrophage ferroptosis in autoimmune diseases

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Studies have also demonstrated that neurons require both the ferroptosis inhibitory factor, GPX4, and genes involved in the GPX4-synthesis pathway to survive under conditions of oxidative stress (21,51,52)